1. Compare the pathophysiology of chronic venous insufficiency and deep venous thrombosis. Describe how venous thrombosis is different from arterial thrombosis.
This particular papers searches for to determine the pathophysiology similarities along with distinctions regarding chronic venous insufficiency and the deep venous thrombosis. Similarities and Differences of Chronic Venous Insufficiency and Deep Venous Thrombosis. Chronic venous insufficiency is impaired venous return, sometimes causing lower extremity edema, skin changes, and discomfort. Causes of chronic venous insufficiency are disorders that lead to venous hypertension, usually by means of venous injury or even incompetence regarding venous valves, because takes place as an example following deep venous thrombosis. Valves inside deep along with perforator blood vessels may possibly in the same manner be generally lacking, letting blood to be able to reflux straight down the actual deep blood vessels, along with outwards on the deep to the superficial blood vessels in the course of leg muscle tissue contraction. While, Deep Venous Thrombosis quite often benefits coming from endothelial damage due to middle venous catheters, pacemakers, or even shot medicine work with. DVT from time to time takes place included in exceptional vena cava, SVC, symptoms or even subclavian train of thought compression setting for the thoracic wall socket. This compression setting maybe due to a regular or even a great accessory 1st rib on a physically demanding supply action. Deep venous thrombosis is actually to begin with paid for by the leg muscle tissue pump, however sooner or later leads to second incompetence regarding valves inside perforating blood vessels, along with transmission regarding demand on the deep to the superficial blood vessels.
Arterial thrombi have a tendency to take place with areas wherever plaques usually are made along with wherever shear anxiety is actually higher, which usually leads to platelet-rich ‘‘white thrombi. ’’ On the other hand, inside venous thrombotic sickness, thrombi have a tendency to take place with sites in which the train of thought wall is actually undamaged, however wherever blood circulation along with shear anxiety usually are small, producing red-colored cell-rich ‘‘red thrombi. ’’ As a result, antiplatelet therapies is considered the favorite choice inside stopping arterial thrombosis, although anticoagulant therapies will be the proposed therapies inside venous thrombosis.
2. Explain how the patient factor you selected might impact the pathophysiology of CVI and DVT. Describe how you would diagnose and prescribe treatment of these disorders and prescribe treatment of these disorders for a patient based on the factor you selected.
Chronic venous insufficiency, CVI, describes more advanced forms of long standing venous dysfunction of the limbs, associated with edema, skin changes, and or active venous ulcerations. While varicose veins affect more than 25% of women and 15% of men in the country, CVI was estimated to occur in up to 3% of the population in European countries, a prevalence comparable to that of diabetes.
While incidence of DVT is between 1% – 6% [per 1000 persons per year]. Prospective studies shows that post-thrombic syndrome developed after DVT in 17% of the limbs at a period of one year.
Diagnosis of DVT is typically by ultrasonography with Doppler flow studies. Treatment of DVT involves anticoagulation with an injectable heparin followed by an oral anticoagulant.
Diagnosis of CVI involves clinical testing and Ultrasonography mainly to exclude DVT. Treatment involves leg elevation; compression using bandages, stockings, and pneumatic devices; topical wound care; and surgery, depending on the disorder’s severity. Some experts believe that weight loss, regular exercise, and reduction of dietary sodium may benefit patients with bilateral chronic venous insufficiency (Huether, S. E., & McCance, K. L., 2012).
3. Construct two mind maps – one for chronic venous insufficiency and one for deep venous thrombosis. Include the epidemiology, pathophysiology, and clinical presentation, as well as the diagnosis and treatment you explained in your paper.
(Zimbron, J., 2008).
Huether, S. E., & McCance, K. L. . (2002). Understanding pathophysiology. St. Louis, MO: Laureate custom ed.
Zimbron, J. (2008). Mind mapsâ€”Dementia, endocarditis, and gastro-oesophageal reflux disease (GERD) [PDF]. Retrieved from http://www.medmaps.co.uk/beta/Gastro-oesophageal reflux disease.
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